Eiichi Kumamoto, Tsugumi Fujita, Rika Suzuki, Nobuya Magori and Chong Wang
Antinociception is produced as a result of various actions including the modulation by analgesics of transient receptor potential (TRP) channel activation and nerve conduction. Elucidation of such a modulation produced by analgesics may serve to understand a cellular mechanism for antinociception.
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